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8600 Old Georgetown RoadBethesda, MD 20814
Phone+1 301-896-3100
Fax+1 301-896-3101
Dr. Simonds is on Doximity
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Education & Training
National Institutes of Health Clinical CenterFellowship, Endocrinology, Diabetes, and Metabolism, 1989 - 1992
MedStar Health Georgetown University/Georgetown HospitalResidency, Internal Medicine, 1985 - 1987
Genesee HospitalInternship, Internal Medicine, 1981 - 1982
University of Pittsburgh School of MedicineClass of 1981
Certifications & Licensure
DC State Medical License 2021 - 2026
MD State Medical License 1988 - 2025
PA State Medical License 2018 - 2018
American Board of Internal Medicine Internal Medicine- American Board of Internal Medicine Endocrinology, Diabetes and Metabolism
Publications & Presentations
PubMed
- 1 citationsGenotype-Phenotype Correlations in the Hyperparathyroidism-Jaw Tumor Syndrome.William F Simonds, Yulong Li, Smita Jha
The Journal of Clinical Endocrinology and Metabolism. 2025-03-17 - Genotype-phenotype correlation in multiple endocrine neoplasia type 1.Charlita C Worthy, Rana Tora, Chandra N Uttarkar, James M Welch, Lynn Bliss
JCI Insight. 2025-02-13 - A Comparative Genomic Analysis of Parathyroid Adenomas and Carcinomas Harboring Heterozygous Germline CDC73 Mutations.Yulong Li, William F Simonds, Haobin Chen
The Journal of Clinical Endocrinology and Metabolism. 2025-01-21
Press Mentions
- Inhibition of G-protein Signalling in Cardiac Dysfunction of Intellectual Developmental Disorder with Cardiac Arrhythmia (IDDCA) SyndromeNovember 9th, 2020
Grant Support
- Mechanism Of Action Of The HRPT2 Tumor Suppressor Gene Product ParafibrominNational Institute Of Diabetes And Digestive And Kidney Diseases2009–2011
- Neuroendocrine Cell Biology Of The G Protein Beta5/ R7-Rgs Protein/ R7BP ComplexNational Institute Of Diabetes And Digestive And Kidney Diseases2009
- Neuroendocrine Cell Biology Of The G Protein Beta5/ R7-Rgs Protein/ R7BP ComplexNational Institute Of Diabetes And Digestive And Kidney Diseases2007–2008
- Mechanism Of G Protein Beta5/ R7-Rgs Protein/ R7BP Complex Signal TransductionNational Institute Of Diabetes And Digestive And Kidney Diseases2007–2008
- Mechanism Of Action Of The HRPT2 Tumor Suppressor Gene Product ParafibrominNational Institute Of Diabetes And Digestive And Kidney Diseases2007–2008
- Clinical Investigation Of Fihp, Parathyroid Cancer &The Hpt-Jt SyndromeNational Institute Of Diabetes And Digestive And Kidney Diseases2007–2008
- Investigation Of Familial Isolated Hpt, Parathyroid CancNational Institute Of Diabetes And Digestive And Kidney Diseases2004–2006
- G Protein Beta-Gamma And Beta-Rgs Dimers--Structure AndNational Institute Of Diabetes And Digestive And Kidney Diseases2001–2006
- G Protein Beta-Gamma And Beta-Rgs Dimers--Structure /FunNational Institute Of Diabetes And Digestive And Kidney Diseases2005
- Familial Isolated Hpt, Parathyroid Cancer, Hpt-Jt SyndroNational Institute Of Diabetes And Digestive And Kidney Diseases2005
- Investigation Of Familial Isolated Hyperparathyroidism ANational Institute Of Diabetes And Digestive And Kidney Diseases2002–2003
- G Protein Beta-Gamma And Beta-Rgs Dimers--Structure And FunctionNational Institute Of Diabetes And Digestive And Kidney Diseases1998–2000
- Guanine Nucleotide Binding Protein Beta-Gamma Dimers--Structure And FunctionNational Institute Of Diabetes And Digestive And Kidney Diseases1994–1997
- Analysis of polygenic disease risk and etiology by indexing ancestry- and gender-specific gene variants predicted to impact functionNATIONAL INSTITUTE OF DIABETES AND DIGESTIVE AND KIDNEY DISEASESPresent
- Analysis of polygenic disease risk and etiology by indexing ancestry- and gender-specific gene variants predicted to impact functionNATIONAL INSTITUTE OF DIABETES AND DIGESTIVE AND KIDNEY DISEASESPresent
- Mechanism of G Protein Beta5/ R7-RGS Protein/ R7BP Complex Signal TransductionNATIONAL INSTITUTE OF DIABETES AND DIGESTIVE AND KIDNEY DISEASESPresent
- Clinical Investigation of FIHP, Parathyroid Cancer & the HPT-JT SyndromeNATIONAL INSTITUTE OF DIABETES AND DIGESTIVE AND KIDNEY DISEASESPresent
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