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Uci Medical Center101 The City Drive SouthOrange, CA 92868
Phone+1 714-456-8978
Dr. Weiss is on Doximity
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Education & Training
Stanford Health Care-Sponsored Stanford UniversityResidency, Neurology, 1984 - 1987- Stanford Health Care-Sponsored Stanford UniversityInternship, Internal Medicine, 1983 - 1984
Stanford University School of MedicineClass of 1983
Certifications & Licensure
CA State Medical License 1984 - 2026
American Board of Psychiatry and Neurology Neurology
Publications & Presentations
PubMed
- 6 citationsBlocking Mitochondrial ZnAccumulation after Ischemia Reduces Mitochondrial Dysfunction and Neuronal Injury.Yuliya V Medvedeva, Hong Z Yin, Afsheen Bazrafkan, Andriy Yeromin, Sung G Ji
The Journal of Neuroscience. 2022-06-29 - 12 citationsVCP/p97 inhibitor CB-5083 modulates muscle pathology in a mouse model of VCP inclusion body myopathy.Cheng Cheng, Lan Weiss, Henri Leinonen, Alyaa Shmara, Hong Z Yin
Journal of Translational Medicine. 2022-01-08 - 24 citationsThe Multifaceted Roles of Zinc in Neuronal Mitochondrial Dysfunction.Hilary Y. Liu, Jenna R. Gale, Ian J. Reynolds, John H. Weiss, Elias Aizenman
Biomedicines. 2021-04-29
Grant Support
- Mitochondrial Zn2+ accumulation and the induction of ischemic neurodegenerationUNIVERSITY OF CALIFORNIA-IRVINE2022–2026
- Mitochondrial Zn2+ accumulation and the induction of ischemic neurodegenerationUNIVERSITY OF CALIFORNIA-IRVINE2022–2026
- Zn2+, Mitochondria And The Induction Of Ischemic NeurodegenerationNational Institute Of Neurological Disorders And Stroke2010–2012
- Ampa/Kainate Receptors, Free Radicals, And Motor Neuron InjuryNational Institute Of Neurological Disorders And Stroke2006–2010
- Ca2+, Zn2+ And Selective Excitotoxic NeurodegenerationNational Institute Of Neurological Disorders And Stroke2000–2002
- Ampa/Kainate Receptors,Free Radicals,Motor Neuron InjuryNational Institute Of Neurological Disorders And Stroke1999–2002
- Zn2+ And Calcium Permeable Ampa/Kainate ChannelsNational Institute On Aging1998–2002
- Ca++ And Excitotoxic Damage Of Cultured Central NeuronsNational Institute Of Neurological Disorders And Stroke1994–1999
- Chronic Neurotoxicity Of BMAA And Non-Nmda AgonistsNational Institute On Aging1994
- CA2+ And Excitotoxic Damage Of Cultured Central NeuronsNational Institute Of Neurological Disorders And Stroke1992–1993
- The Chronic Neurotoxicity Of BMAA And Non-Nmda AgonistsNational Institute On Aging1991–1993
- Chronic Neurotoxicity Of BMAA And NON NMDA AgonistsNational Institute On Aging1992
- Chronic Neurotoxicity Of BMAA And NON NMDA AgonistsNational Institute On Aging1990–1991
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