Dr. Titterington is on Doximity
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Office
4000 Cambridge St
Ste G600
Kansas City, KS 66160Phone+1 913-588-9700Fax+1 913-588-9770
Education & Training
- Emory University School of MedicineFellowship, Advanced Heart Failure and Transplant Cardiology, 2017 - 2018
- Emory University School of MedicineFellowship, Cardiovascular Disease, 2012 - 2017
- Emory University School of MedicineResidency, Internal Medicine, 2010 - 2013
- Tulane University School of MedicineClass of 2010
- Kansas State UniversityB.A., Microbiology major, Spanish minor, 1998 - 2002
Certifications & Licensure
- KS State Medical License 2018 - 2023
- MO State Medical License 2018 - 2023
- GA State Medical License Active through 2018
Awards, Honors, & Recognition
- Haymaker Award Kansas State University Division of Biology, 2002
- Phi Beta Kappa 2002
- Research Award Kansas State University Center for Basic Cancer, 2001
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Publications & Presentations
PubMed
- 202 citationsIGF-1 Reduces Inflammatory Responses, Suppresses, Oxidative Stress and Decreases Atherosclerosis Progression in Apoe-Deficient MiceSergiy Sukhanov, Yusuke Higashi, Shaw-Yung Shai, Charlotte Vaughn, Jessica Mohler
Arteriosclerosis, Thrombosis, and Vascular Biology. 2007-12-01 - 9 citationsMicrovascular angina: an update on diagnosis and treatment.Jane S Titterington, Olivia Y. Hung, Nanette K. Wenger
Future Cardiology. 2015-03-11 - 4 citationsGender differences in acute coronary syndromes: focus on the women with ACS without an obstructing culprit lesion.Jane S Titterington, Olivia Y. Hung, Anita Saraf, Nanette K. Wenger
Expert Review of Cardiovascular Therapy. 2018-03-07
Abstracts/Posters
- Growth hormone-releasing peptide-2 reduces oxidative stress in ApoE -/- mice but does not reduce atherosclerosis.Titterington JS, Sukhanov S, Vaughn CE, Delafontaine P, FASEB J, 1/1/2009
- Insulin-like growth factor I downregulates lipoprotein lipase and suppresses atherosclerotic foam cell formation in vivo and in vitro.Sukhanov S. Vaughn CE, Higashi Y, Titterington JS, Delafontaine P, FASEB J, 1/1/2009
- Chronic Left-Ventricular Dysfunction Increases Oxidative Stress in Skeletal Muscle: Potential Mechanism of Cardiac Cachexia.Semprun-Prieto L, Sukhanov S, Bhutto J, Titterington JS, Delafontaine P, FASEB J, 1/1/2009
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Lectures
- Growth hormone-releasing peptide-2 reduces oxidative stress in ApoE -/- mice but does not reduce atherosclerosis.Experimental Biology - 4/1/2009
- Insulin-like growth factor I downregulates lipoprotein lipase and suppresses atherosclerotic foam cell formation in vivo and in vitro.Experimental Biology - 4/1/2009
- Chronic Left-Ventricular Dysfunction Increases Oxidative Stress in Skeletal Muscle: Potential Mechanism of Cardiac Cachexia.Experimental Biology - 4/1/2009
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Press Mentions
- Resveratrol-Mediated Expression of KLF15 in the Ischemic Myocardium Is Associated with an Improved Cardiac PhenotypeJanuary 7th, 2017
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