Dr. Titterington is on Doximity
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Office
4000 Cambridge St
Ste G600
Kansas City, KS 66160Phone+1 913-588-9700Fax+1 913-588-9770
Education & Training
- Emory University School of MedicineFellowship, Advanced Heart Failure and Transplant Cardiology, 2017 - 2018
- Emory University School of MedicineFellowship, Cardiovascular Disease, 2012 - 2017
- Emory University School of MedicineResidency, Internal Medicine, 2010 - 2013
- Tulane University School of MedicineClass of 2010
- Kansas State UniversityB.A., Microbiology major, Spanish minor, 1998 - 2002
Certifications & Licensure
- KS State Medical License 2018 - 2023
- MO State Medical License 2018 - 2023
- GA State Medical License Active through 2018
Awards, Honors, & Recognition
- Haymaker Award Kansas State University Division of Biology, 2002
- Phi Beta Kappa 2002
- Research Award Kansas State University Center for Basic Cancer, 2001
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Publications & Presentations
PubMed
- 23 citationsNovel PET and Near Infrared Imaging Probes for the Specific Detection of Bacterial Infections Associated With Cardiac Devices.Kiyoko Takemiya, Xinghai Ning, Wonewoo Seo, Xiaojian Wang, Rafi Mohammad
JACC. Cardiovascular Imaging. 2019-05-01 - 4 citationsGender differences in acute coronary syndromes: focus on the women with ACS without an obstructing culprit lesion.Jane S Titterington, Olivia Y. Hung, Anita Saraf, Nanette K. Wenger
Expert Review of Cardiovascular Therapy. 2018-03-07 - 2 citationsEvolving cardiovascular care for women: a decade of progressOlivia Y. Hung, Jane S Titterington, Nanette K. Wenger
Future Cardiology. 2015-05-29
Abstracts/Posters
- Growth hormone-releasing peptide-2 reduces oxidative stress in ApoE -/- mice but does not reduce atherosclerosis.Titterington JS, Sukhanov S, Vaughn CE, Delafontaine P, FASEB J, 1/1/2009
- Insulin-like growth factor I downregulates lipoprotein lipase and suppresses atherosclerotic foam cell formation in vivo and in vitro.Sukhanov S. Vaughn CE, Higashi Y, Titterington JS, Delafontaine P, FASEB J, 1/1/2009
- Chronic Left-Ventricular Dysfunction Increases Oxidative Stress in Skeletal Muscle: Potential Mechanism of Cardiac Cachexia.Semprun-Prieto L, Sukhanov S, Bhutto J, Titterington JS, Delafontaine P, FASEB J, 1/1/2009
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Lectures
- Growth hormone-releasing peptide-2 reduces oxidative stress in ApoE -/- mice but does not reduce atherosclerosis.Experimental Biology - 4/1/2009
- Insulin-like growth factor I downregulates lipoprotein lipase and suppresses atherosclerotic foam cell formation in vivo and in vitro.Experimental Biology - 4/1/2009
- Chronic Left-Ventricular Dysfunction Increases Oxidative Stress in Skeletal Muscle: Potential Mechanism of Cardiac Cachexia.Experimental Biology - 4/1/2009
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Press Mentions
- Resveratrol-Mediated Expression of KLF15 in the Ischemic Myocardium Is Associated with an Improved Cardiac PhenotypeJanuary 7th, 2017
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