ErnestoBernal-MizrachiMD
Endocrinology • Coral Gables, FLAdrenal Disease, Diabetes, Lipid Metabolism, Pituitary Disorders & Neuroendocrinology
Chief, Division of Endocrinology, Diabetes and Metabolism, University of Miami Miller School of Medicine
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Office
5555 Ponce de Leon Blvd
Coral Gables, FL 33146Phone+1 305-689-3636Fax+1 305-243-6575
Education & Training
Washington University/B-JH/SLCH ConsortiumFellowship, Endocrinology, Diabetes, and Metabolism, 1996 - 1999
University of Miami/Jackson Health SystemResidency, Internal Medicine, 1993 - 1996
Universidad del Valle Health SciencesClass of 1989
Certifications & Licensure
FL State Medical License 2016 - 2026
MI State Medical License 2009 - 2019
MO State Medical License 1999 - 2010
American Board of Internal Medicine Endocrinology, Diabetes and Metabolism
Awards, Honors, & Recognition
- Elected Member The American Society for Clinical Investigation, 2010
Publications & Presentations
PubMed
- L-type calcium channel blockade worsens glucose tolerance and β-cell function in C57BL6/J mice exposed to intermittent hypoxia.Stanley M Chen Cardenas, Tess A Baker, Larissa A Shimoda, Ernesto Bernal-Mizrachi, Naresh M Punjabi
American Journal of Physiology. Endocrinology and Metabolism. 2025-02-01 - Glycemic Control in Patients with Employer-Sponsored Health Benefits.Judy Z Louie, Charles M Rowland, Dov Shiffman, Rajesh Garg, Ernesto Bernal-Mizrachi
Population Health Management. 2025-02-01 - L-type Calcium Channel Blockade Worsens Glucose Tolerance and β-Cell Function in C57BL6/J Mice Exposed to Intermittent Hypoxia.Stanley M Chen-Cardenas, Tess A Baker, Larissa A Shimoda, Ernesto Bernal-Mizrachi, Naresh M Punjabi
American Journal of Physiology. Endocrinology and Metabolism. 2025-01-10
Press Mentions
Research Illuminates Potential Pathway to Type 2 DiabetesSeptember 21st, 2017- Enzyme Protects Insulin-Producing Cells Against Chronic High GlucoseJanuary 12th, 2016
Grant Support
- Loss of insulin signaling across functional pancreas compartments as a major pathogenic mechanism underlying diabetic exocrine pancreatopathyUNIVERSITY OF MIAMI SCHOOL OF MEDICINE2024–2028
- Loss of insulin signaling across functional pancreas compartments as a major pathogenic mechanism underlying diabetic exocrine pancreatopathyUNIVERSITY OF MIAMI SCHOOL OF MEDICINE2024–2028
- mTOR signaling and regulation of alpha-cell mass and functionMIAMI VA HEALTH CARE SYSTEM2016–2028
- mTOR signaling and regulation of alpha-cell mass and functionMIAMI VA HEALTH CARE SYSTEM2016–2028
- mTOR signaling and regulation of alpha-cell mass and functionMIAMI VA HEALTH CARE SYSTEM2016–2028
- mTOR signaling and regulation of alpha-cell mass and functionMIAMI VA HEALTH CARE SYSTEM2016–2028
- mTOR signaling and regulation of alpha-cell mass and functionMIAMI VA HEALTH CARE SYSTEM2016–2028
- Amino acid sensing mechanisms in beta and alpha cellsUNIVERSITY OF MIAMI SCHOOL OF MEDICINE2022–2027
- Role of mTORC1 signaling in type 1 diabetesUNIVERSITY OF MIAMI SCHOOL OF MEDICINE2022–2026
- Nutrient Signals And Programming Of Pancreas DevelopmentNational Institute Of Diabetes And Digestive And Kidney Diseases2010–2011
- Akt/Mtor Signaling And Regulation Of Cell Cycle In Beta CellsNational Institute Of Diabetes And Digestive And Kidney Diseases2009–2010
- Akt/Mtor Signaling And Regulation Of Cell Cycle In Beta CellsNational Institute Of Diabetes And Digestive And Kidney Diseases2006–2009
- Regulation Of Pancreas Development By P13k/Akt SignalingNational Institute Of Diabetes And Digestive And Kidney Diseases2004–2006
Professional Memberships
- Member
- Member
Other Languages
- Spanish
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