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Edward Lakatta, MD, Cardiology, Baltimore, MD

EdwardGerardLakattaMD

Cardiology • Baltimore, MD

Adult Congenital Heart Disease

Adjunct Professor, Physiology, University of Maryland School of Medicine

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  • Office

    5600 Nathan Shock Dr
    Baltimore, MD 21224
    Phone+1 410-558-8202
    Fax+1 410-558-8150

Education & Training

  • Johns Hopkins University
    Johns Hopkins UniversityResidency, Internal Medicine, 1975 - 1976
  • MedStar Health/Georgetown-Washington Hospital Center
    MedStar Health/Georgetown-Washington Hospital CenterFellowship, Cardiovascular Disease, 1974 - 1975
  • University of Rochester
    University of RochesterResidency, Internal Medicine, 1970 - 1972
  • Georgetown University School of Medicine
    Georgetown University School of MedicineClass of 1970

Certifications & Licensure

  • MD State Medical License
    MD State Medical License 1973 - 2026
  • American Board of Internal Medicine Internal Medicine
  • American Board of Internal Medicine Cardiovascular Disease

Awards, Honors, & Recognition

  • Elected Member The American Society for Clinical Investigation, 1984

Clinical Trials

Publications & Presentations

PubMed

Journal Articles

  • The Dilemma of Coronavirus Disease 2019, Aging, and Cardiovascular Disease  
    Majd Alghatrif, Oscar Cingolani, Edward G Lakatta, JAMA Cardiology

Authored Content

  • The Dilemma of Coronavirus Disease 2019, Aging, and Cardiovascular DiseaseMay 2020
  • The Dilemma of Coronavirus Disease 2019, Aging, and Cardiovascular DiseaseApril 2020
  • The Dilemma of Coronavirus Disease 2019, Aging, and Cardiovascular DiseaseApril 2020

Grant Support

  • Mitochondrial-Sr CA2+ Cycling Interaction In Cardiac Pacemaker CellsNational Institute On Aging2011
  • Beat To Beat Ca2+-Dependent Regulation Of Pacemaker Cell Rate And RhythmNational Institute On Aging2011
  • Adenylyl Cyclase And Phosphodiesterase In Lipid Rafts Of Pacemaker CellsNational Institute On Aging2011
  • The Reveal StudyNational Institute On Aging2010–2011
  • Protein Phosphatase1 Regulates Normal Automaticity Of Heart Pacemaker Node CellsNational Institute On Aging2010–2011
  • Biochemistry And Signaling Of Receptor For Advanced Glycation END ProductsNational Institute On Aging2010–2011
  • Are Sanc From The Center Or Periperal Area Of The Sinoatrial Node Different?National Institute On Aging2010–2011
  • Soluble Receptor For Advanced Glycation END Products For Therapeutic ApplicationNational Institute On Aging2009–2011
  • Phosphodiesterases Restrict Spontaneous Beating Of Cardiac Pacemaker CellsNational Institute On Aging2009–2011
  • Phosphodiesterases 3 And 4 Regulate Local CA2+ Releases And Beating Of PacemakerNational Institute On Aging2009–2011
  • Matching ATP Supply And Demand In Cardiac Pacemaker CellsNational Institute On Aging2009–2011
  • Genome-Wide Association Study Of Cardiovascular Traits In 6142 SardiniansNational Institute On Aging2009–2011
  • Effects Of Aging On Experimental Atherosclerosis In Nonhuman PrimatesNational Institute On Aging2009–2011
  • Effects Of Age And Conditioning Status On REST And Exercise Cardiac PerformanceNational Institute On Aging2009–2011
  • Effect On Metalloprotease Inhibition On Age-Associated Arterial RemodelingNational Institute On Aging2009–2011
  • Decreased Pacemaker Activity In Aged Sinoatrial NodeNational Institute On Aging2009–2011
  • Cultured Adult Rabbit Pacemaker Cells For Gene Transfer StudiesNational Institute On Aging2009–2011
  • Calpain-1 Activity And Central Arterial AgingNational Institute On Aging2009–2011
  • Both PKA And Camkii Phosphorylation Drive Pacemaker Cell AutomaticityNational Institute On Aging2009–2011
  • Age-Associated Changes In Arterial Proteome And Aortic Smooth Muscle SignalingNational Institute On Aging2009–2011
  • Age Associated Changes In Structural And Functional Cardio-Vascular PropertiesNational Institute On Aging2009–2011
  • A PUFA Dietary Intervention For Heart RateNational Institute On Aging2009–2011
  • Suppression Of Beta-Arrestin1 Phosphorylation And Function By Beta-Arrestin2National Institute On Aging2010
  • RGS2 Constitutes A Negative Regulator Of Beta2-Adrenergic GI SignalingNational Institute On Aging2010
  • Agonist Stereochemistry Determines Beta2-Adrenergic G Protein CouplingNational Institute On Aging2010
  • Dilated Cardiomyopathy With Increased SR CA2+ Loading In The Alpha 1ctg MouseNational Institute On Aging2009–2010
  • Cholinergic Regulation Of Pka-Dependent CA2+ Cycling In Pacemaker CellsNational Institute On Aging2009–2010
  • Ca2+-Stimulated Basal Adenylyl Cyclase Activity Localization In Membrane LipidNational Institute On Aging2009–2010
  • Phosphodiesterases Restrict Spontaneous Beating Of Cardiac Pacemaker CellsNational Institute On Aging2008
  • Phosphodiesterases 3 And 4 Regulate Local CA2+ Releases And Beating Of PacemakerNational Institute On Aging2008
  • Effects Of Aging On Experimental Atherosclerosis In Nonhuman PrimatesNational Institute On Aging2008
  • Effect On Metalloprotease Inhibition On Age-Associated Arterial RemodelingNational Institute On Aging2008
  • Dilated Cardiomyopathy With Increased SR CA2+ Loading In The Alpha 1ctg MouseNational Institute On Aging2008
  • Defining Unique Cardiac Proteome: Comparing Sino-Atrial Node, Atria, VentricleNational Institute On Aging2008
  • Cholinergic Regulation Of Pka-Dependent CA2+ Cycling In Pacemaker CellsNational Institute On Aging2008
  • Calpain-1 Activity And Central Arterial AgingNational Institute On Aging2008
  • Both PKA And Camkii Phosphorylation Drive Pacemaker Cell AutomaticityNational Institute On Aging2008
  • Age-Associated Changes In Arterial Proteome And Aortic Smooth Muscle SignalingNational Institute On Aging2008
  • Age Associated Changes In Structural And Functional Cardio-Vascular PropertiesNational Institute On Aging2007–2008
  • Progress In The Intracellular Clock That Drives The Heart'S PacemakerNational Institute On Aging2007
  • Central Arterial Aging: Humans To MoleculesNational Institute On Aging2006–2007
  • Progress In The Intracellular Clock That Drives The HearNational Institute On Aging2006
  • Age Associated Changes In Vascular Stiffness PropertiesNational Institute On Aging1992–2002
  • Activation Of Distinct Camp- And Cgmp-Dependent Pathways By NO In CardiomyocytesNational Institute On Aging1998–2000
  • Ion Transport MechanismsNational Institute On Aging1999
  • Mechanism For Signal Transduction Of Opioid Receptor Stimulation Of MyocytesNational Institute On Aging1992–1993
  • Age Associated Changes In Vascular PropertiesNational Institute On Aging1991

Dr. Edward Lakatta, Dr. Edward Lakatta, MD, Dr. E Lakatta, Dr. Edward Gerard Lakatta