DavidRCroweMD
Pathology • Birmingham, ALCytopathology, Pulmonary Pathology, Anatomic Pathology, Clinical Pathology
Professor of Pathology, University of Alabama School of Medicine
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Office
619 19th St S
Birmingham, AL 35233Phone+1 205-934-6600Fax+1 205-975-7284
Summary
- Dr. David Crowe is a pathologist in Birmingham, AL and is affiliated with multiple hospitals in the area, including UAB Highlands, UAB Hospital, Birmingham Veterans Affairs Medical Center, and Central Alabama Veterans Health Care System. He received his medical degree from University of Alabama School of Medicine and has been in practice 40 years. He specializes in cytopathology, pulmonary pathology, anatomic pathology, and clinical pathology and is experienced in pulmonary and liver transplant pathology.
Dr, Crowe retired from medical practice on 12/0102021.
Education & Training
University of Alabama Medical CenterResidency, Pathology-Anatomic and Clinical, 1979 - 1981
Physicians Medical Center CarrawayResidency, Pathology-Anatomic and Clinical, 1977 - 1979
University of Alabama School of MedicineClass of 1977
Certifications & Licensure
AL State Medical License 1978 - 2025- American Board of Pathology Anatomic Pathology & Clinical Pathology
Publications & Presentations
PubMed
- 1 citationsEvaluating the consistency with guideline recommendations for diagnosis and management of idiopathic pulmonary fibrosis in non-academic settings.Joao De Andrade, Tracy Luckhardt, Sushilkumar Sonavane, D Ralph Crowe, Tejaswini Kulkarni
Sarcoidosis, Vasculitis, and Diffuse Lung Diseases. 2023-03-28 - 5 citationsClub Cell Heme Oxygenase-1 Deletion: Effects in Hyperoxia-Exposed Adult Mice.Katelyn Dunigan-Russell, Mary Silverberg, Vivian Y Lin, Rui Li, Stephanie B. Wall
Oxidative Medicine and Cellular Longevity. 2020-06-09 - 16 citationsExtracellular nucleic acid scavenging rescues rats from sulfur mustard analog-induced lung injury and mortalityNithya Mariappan, Maroof Husain, Iram Zafar, V. Singh, Kenneth G. Smithson
Archives of Toxicology. 2020-03-10
Press Mentions
Modulation of H4K16Ac Levels Reduces Pro-Fibrotic Gene Expression and Mitigates Lung Fibrosis in Aged MiceMay 14th, 2021
Professional Memberships
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