Dr. Haber is on Doximity
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Office
55 Fruit St
149 7202
Boston, MA 02114Phone+1 617-726-7805Fax+1 617-724-6919
Education & Training
Massachusetts Institute of TechnologyPost-Doctoral Fellowship, 1987 - 1991
Brigham and Women's Hospital/Massachusetts General Hospital/Dana-Farber Cancer InstituteFellowship, Hematology and Medical Oncology, 1986 - 1990
Massachusetts General HospitalResidency, Internal Medicine, 1984 - 1986- Massachusetts General HospitalInternship, Internal Medicine, 1983 - 1984
Stanford University School of MedicineClass of 1983
Certifications & Licensure
MA State Medical License 1985 - 2025- American Board of Internal Medicine Internal Medicine
- American Board of Internal Medicine Medical Oncology
Awards, Honors, & Recognition
- Elected Member Fellow of the AACR Academy, 2019
- Elected Member National Academy of Sciences, 2018
- Investigator Howard Hughes Medical Institute
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Publications & Presentations
PubMed
- 121 citationsThe Tre2 (USP6) oncogene is a hominoid-specific geneCharles Paulding, Maryellen Ruvolo, Daniel A. Haber
Proceedings of the National Academy of Sciences of the United States of America. 2003-03-04 - 383 citationsInherited susceptibility to lung cancer may be associated with the T790M drug resistance mutation in EGFRDaphne W. Bell, Ira Gore, Ross A. Okimoto, Godin-Heymann Nadia G, Raffaella Sordella
Nature Genetics. 2005-10-30 - 111 citationsInactivation of WT1 in nephrogenic rests, genetic precursors to Wilms' tumourSo Yeon Park, Amy Bernard, K E Bove, Donald A. Sens, Debra J. Hazen-Martin
Nature Genetics. 1993-12-01
Journal Articles
- A Digital RNA Signature of Circulating Tumor Cells Predicting Early Therapeutic Response in Localized and Metastatic Breast CancerDavid T Miyamoto, Aditya Bardia, Lecia V Sequist, Laura M Spring, Daniel A Haber, Mark Kalinich, Clinical Cancer Research
Press Mentions
Isolation of Circulating Tumor Cells with TellDx Platform Reveals Potential Mechanism of Resistance to Immune Checkpoint Blockade in MelanomaMarch 16th, 2023- Reduction in Circulating Tumor Cells (CTC), Enriched by TellBio Platform, with Novel Dual-Cadherin Antibody (23C6) Results in Significant Metastasis SuppressionOctober 24th, 2022
- TellBio’s Scientific Founders Publish Very High Concordance Between CTCs and ctDNA from Liquid Biopsies in Patients with Breast CancerJuly 15th, 2021
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Grant Support
- Circumventing Acquired Resistance To Growth Factor Receptor Kinase InhibitorsNational Cancer Institute2008–2012
- Data Production, And Informatics And IntegrationNational Human Genome Research Institute2010–2011
- P1 - Clinical Correlations Of WTX Inactivation In Wilms TumorNational Cancer Institute2010–2011
- Functional Properties Of The Wilms'Tumor Gene WT1National Cancer Institute2008–2011
- Functional Properties Of The Wilms' Tumor Gene WT1National Cancer Institute2010
- Clinical Correlations Of WTX Inactivation In Wilms TumorNational Cancer Institute2009
- Functional Properties Of The Wilm'S Tumor Gene WT1National Cancer Institute2003–2007
- Targets Of Ewing Sarcoma-Wilms Tumor 1 OncoproteinNational Cancer Institute2002–2006
- Genetic Models Of Cellular ProliferationNational Cancer Institute2002–2006
- Functional Characterization Of BRCA1National Cancer Institute2000–2004
- Screen For Genes Targeted By Homozygous Deletion During Cancer ProgressionNational Cancer Institute2002
- G2 Checkpoint Genes In Breast Cancer SusceptibiityNational Cancer Institute2000–2002
- Functional Properties Of The Wilms Tumor Gene WT1National Cancer Institute1998–2002
- Mutational Analysis Of P53 Related GenesNational Cancer Institute2000–2001
- Isolation Of Gene For X-Linked LymphoproliferationNational Cancer Institute1995–1998
- Mutational And Functional Analysis Of Wilm'S TumorNational Cancer Institute1994–1997
- Mutational And Functional Analysis Of Wilms'TumorNational Cancer Institute1993
- Multidrug Resistance Genes--Expression And FunctionNational Cancer Institute1990
- Multidrug Resistance Genes--Expression And FunctionNational Cancer Institute1988–1989
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