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Bradley Hyman, MD, Neurology, Boston, MA

BradleyTHymanMDPhD

Neurology Boston, MA

Neurodegenerative

John B. Penney Professor of Neurology Harvard Medical School Alzheimer's Unit Director MassGeneral Institute for Neurodegenerative Disease Director Massachusetts Alzheimer's Disease Research Center

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  • Office

    15 Parkman St
    Wac 835
    Boston, MA 02114
    Phone+1 617-726-1728
    Fax+1 617-726-2894

Education & Training

  • University of Iowa Hospitals and Clinics
    University of Iowa Hospitals and ClinicsFellowship, Neuropathology, 1988 - 1989
  • University of Iowa Hospitals and Clinics
    University of Iowa Hospitals and ClinicsResidency, Neurology, 1985 - 1988
  • University of Iowa Hospitals and Clinics
    University of Iowa Hospitals and ClinicsInternship, Internal Medicine, 1984 - 1985
  • University of Iowa Roy J. and Lucille A. Carver College of Medicine
    University of Iowa Roy J. and Lucille A. Carver College of MedicineClass of 1983

Certifications & Licensure

  • ME State Medical License
    ME State Medical License 2021 - 2026
  • MA State Medical License
    MA State Medical License 1989 - 2025
  • NH State Medical License
    NH State Medical License 2021 - 2025
  • IA State Medical License
    IA State Medical License 1984 - 1990
  • American Board of Psychiatry and Neurology Neurology

Publications & Presentations

PubMed

Journal Articles

  • Dissecting the Genetic Relationship Between Cardiovascular Risk Factors and Alzheimer’s Disease  
    Christine M Glastonbury, Fanny M Elahi, Reisa A Sperling, William P Dillon, Lindsay A Farrer, Rahul S Desikan, Christopher P Hess, James B Brewer, Bradley T Hyman, Kri..., Acta Neuropathologica
  • Absence of Alzheimer Disease Neuropathologic Changes in Eyes of Subjects with Alzheimer Disease  
    Bradley T. Hyman, Nora Laver, Declan McGuone, Journal of Neuropathology & Experimental Neurology

Press Mentions

  • A Novel Age-Informed Approach for Genetic Association Analysis in Alzheimer’s Disease
    A Novel Age-Informed Approach for Genetic Association Analysis in Alzheimer’s DiseaseApril 1st, 2021
  • Does Astrocyte Tau Cause Dementia?
    Does Astrocyte Tau Cause Dementia?November 13th, 2020
  • Decline in Cognitively Complex Everyday Activities Accelerates Along the Alzheimer’s Disease Continuum
    Decline in Cognitively Complex Everyday Activities Accelerates Along the Alzheimer’s Disease ContinuumOctober 29th, 2020
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Grant Support

  • Calcineurin-Mediated Neurodegeneration In Alzheimer DiseaseNational Institute On Aging2011–2012
  • A Model Of Early Alzheimer DiseaseNational Institute On Aging2011–2012
  • Neuropathological Correlates Of Oligomeric Amyloid BetaNational Institute On Aging2009–2012
  • AdministrativeNational Institute On Aging2009–2012
  • Massachusetts Alzheimer'S Disease Research CenterNational Institute On Aging2006–2012
  • Anatomical Changes In TAU Transgenic ModelsNational Institute On Aging2010–2011
  • Peripherally Administered Gene Therapy For Alzheimer'SNational Institute On Aging2009–2011
  • LRP And Alzheimer'S DiseaseNational Institute On Aging2000–2011
  • Faculty Recruitment For Programs In The Biology Of Age-Related Cognitive ChangeNational Institute On Aging2009–2010
  • Neurologic Alterations In Alzheimer'S DiseaseNational Institute On Aging2006–2010
  • Anatomical Changes In TAU Trangenic ModelsNational Institute On Aging2004–2010
  • Core--ScientificNational Institute Of Neurological Disorders And Stroke2008
  • TAU Alterations In Alzheimer'S And Related DementiasNational Institute On Aging2004–2008
  • Molecular Chaperones In Lewy Body DiseasesNational Institute Of Neurological Disorders And Stroke2004–2008
  • Gamma Secretase And Substrate InteractionsNational Institute On Aging2007
  • Core--ScientificNational Institute Of Neurological Disorders And Stroke2004–2007
  • Neuroanatomical Alterations In Alzheimer'S DiseaseNational Institute On Aging2001–2005
  • Core--NeuropathologyNational Institute On Aging2002
  • Genetic Modifiers Of Amyloid Beta Deposition--Neuroanatomic CorrelatesNational Institute On Aging1999–2002
  • APOE And Transgenic Models Of Alzheimers DiseaseNational Institute On Aging1999–2002
  • Transgenics And Presenilin FunctionNational Institute On Aging1998–2002
  • Role Of Alpha Synuclein In Lewy Body DiseaseNational Institute Of Neurological Disorders And Stroke1998–2002
  • Presenilins And Alzheimers DiseaseNational Institute On Aging1997–1999
  • Neuropathological Alterations In Alzheimers DiseaseNational Institute On Aging1995–1999
  • Signature Of Regional And Neuronal Vulnerability In ADNational Institute On Aging1996–1998
  • Core--Brain Microdissection, RNA And Protein AnalysisNational Institute On Aging1996–1998
  • Structural And Molecular Correlates Of Cognitive Function In Alzheimer'S DiseaseNational Institute On Aging1994–1998
  • Apolipoprotein E And Alzheimers DiseaseNational Institute On Aging1994–1998
  • Pathological Alterations In Alzheimer'S DiseaseNational Institute On Aging1989–1993