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Bradley Hyman, MD, Neurology, Boston, MA

Bradley T Hyman MD PhD

Neurodegenerative


John B. Penney Professor of Neurology Harvard Medical School Alzheimer's Unit Director MassGeneral Institute for Neurodegenerative Disease Director Massachusetts Alzheimer's Disease Research Center

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  • 15 Parkman StWac 835Boston, MA 02114

  • Phone+1 617-726-1728

  • Fax+1 617-726-2894

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Education & Training

  • University of Iowa Hospitals and Clinics
    University of Iowa Hospitals and ClinicsFellowship, Neuropathology, 1988 - 1989
  • University of Iowa Hospitals and Clinics
    University of Iowa Hospitals and ClinicsResidency, Neurology, 1985 - 1988
  • University of Iowa Hospitals and Clinics
    University of Iowa Hospitals and ClinicsInternship, Internal Medicine, 1984 - 1985
  • University of Iowa Roy J. and Lucille A. Carver College of Medicine
    University of Iowa Roy J. and Lucille A. Carver College of MedicineClass of 1983

Certifications & Licensure

  • ME State Medical License
    ME State Medical License 2021 - 2026
  • MA State Medical License
    MA State Medical License 1989 - 2025
  • NH State Medical License
    NH State Medical License 2021 - 2025
  • IA State Medical License
    IA State Medical License 1984 - 1990
  • Neurology
    American Board of Psychiatry and Neurology Neurology

Publications & Presentations

PubMed

Journal Articles

  • Dissecting the Genetic Relationship Between Cardiovascular Risk Factors and Alzheimer’s Disease  
    Christine M Glastonbury, Fanny M Elahi, Reisa A Sperling, William P Dillon, Lindsay A Farrer, Rahul S Desikan, Christopher P Hess, James B Brewer, Bradley T Hyman, Kri..., Acta Neuropathologica
  • Absence of Alzheimer Disease Neuropathologic Changes in Eyes of Subjects with Alzheimer Disease  
    Bradley T. Hyman, Nora Laver, Declan McGuone, Journal of Neuropathology & Experimental Neurology

Press Mentions

  • A Novel Age-Informed Approach for Genetic Association Analysis in Alzheimer’s Disease
    A Novel Age-Informed Approach for Genetic Association Analysis in Alzheimer’s DiseaseApril 1st, 2021
  • Does Astrocyte Tau Cause Dementia?
    Does Astrocyte Tau Cause Dementia?November 13th, 2020
  • Decline in Cognitively Complex Everyday Activities Accelerates Along the Alzheimer’s Disease Continuum
    Decline in Cognitively Complex Everyday Activities Accelerates Along the Alzheimer’s Disease ContinuumOctober 29th, 2020
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Grant Support

  • Discovery and optimization of ApoE4 correctors for the treatment of Alzheimer's DiseaseSANFORD BURNHAM PREBYS MEDICAL DISCOVERY INSTITUTE2024–2029
  • MGH Diseases of Aging Pathway Via Stimulating Access to Research in Residency (MGH DAP StARR)MASSACHUSETTS GENERAL HOSPITAL2023–2027
  • MGH Diseases of Aging Pathway Via Stimulating Access to Research in Residency (MGH DAP StARR)MASSACHUSETTS GENERAL HOSPITAL2023–2027
  • LRP1-tau interactions and Alzheimer DiseaseUNIVERSITY OF MARYLAND BALTIMORE2021–2026
  • Discovery and development of apoE4 correctors for the treatment of Alzheimer's diseaseSANFORD BURNHAM PREBYS MEDICAL DISCOVERY INSTITUTE2023–2025
  • Calcineurin-Mediated Neurodegeneration In Alzheimer DiseaseNational Institute On Aging2011–2012
  • A Model Of Early Alzheimer DiseaseNational Institute On Aging2011–2012
  • Neuropathological Correlates Of Oligomeric Amyloid BetaNational Institute On Aging2009–2012
  • AdministrativeNational Institute On Aging2009–2012
  • Massachusetts Alzheimer'S Disease Research CenterNational Institute On Aging2006–2012
  • Anatomical Changes In TAU Transgenic ModelsNational Institute On Aging2010–2011
  • Peripherally Administered Gene Therapy For Alzheimer'SNational Institute On Aging2009–2011
  • LRP And Alzheimer'S DiseaseNational Institute On Aging2000–2011
  • Faculty Recruitment For Programs In The Biology Of Age-Related Cognitive ChangeNational Institute On Aging2009–2010
  • Neurologic Alterations In Alzheimer'S DiseaseNational Institute On Aging2006–2010
  • Anatomical Changes In TAU Trangenic ModelsNational Institute On Aging2004–2010
  • Core--ScientificNational Institute Of Neurological Disorders And Stroke2008
  • TAU Alterations In Alzheimer'S And Related DementiasNational Institute On Aging2004–2008
  • Molecular Chaperones In Lewy Body DiseasesNational Institute Of Neurological Disorders And Stroke2004–2008
  • Gamma Secretase And Substrate InteractionsNational Institute On Aging2007
  • Core--ScientificNational Institute Of Neurological Disorders And Stroke2004–2007
  • Neuroanatomical Alterations In Alzheimer'S DiseaseNational Institute On Aging2001–2005
  • Core--NeuropathologyNational Institute On Aging2002
  • Genetic Modifiers Of Amyloid Beta Deposition--Neuroanatomic CorrelatesNational Institute On Aging1999–2002
  • APOE And Transgenic Models Of Alzheimers DiseaseNational Institute On Aging1999–2002
  • Transgenics And Presenilin FunctionNational Institute On Aging1998–2002
  • Role Of Alpha Synuclein In Lewy Body DiseaseNational Institute Of Neurological Disorders And Stroke1998–2002
  • Presenilins And Alzheimers DiseaseNational Institute On Aging1997–1999
  • Neuropathological Alterations In Alzheimers DiseaseNational Institute On Aging1995–1999
  • Signature Of Regional And Neuronal Vulnerability In ADNational Institute On Aging1996–1998
  • Core--Brain Microdissection, RNA And Protein AnalysisNational Institute On Aging1996–1998
  • Structural And Molecular Correlates Of Cognitive Function In Alzheimer'S DiseaseNational Institute On Aging1994–1998
  • Apolipoprotein E And Alzheimers DiseaseNational Institute On Aging1994–1998
  • Pathological Alterations In Alzheimer'S DiseaseNational Institute On Aging1989–1993

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