AnkurSethMDMS

Hospital Medicine/Hospitalist

Interim Vice Chair Department of Internal Medicine at UTHSC. Director of the UTHSC Hospitalist training program at Baptist Memorial Hospital.

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Office
Baptist Memorial Hospital
6025 Walnut Grove Rd
Memphis, TN 38120

Summary

Dr. Ankur Seth is an internist in Memphis, TN and is affiliated with multiple hospitals in the area, including Baptist Memorial Hospital - Memphis, University of Tennessee Health Science Center, Baptist Memorial Restorative Care Hospital, and Baptist Memorial Hospital for Women. He received his medical degree from Dayanand Medical College and has been in practice 18 years. He specializes in hospital medicine/hospitalist and is experienced in clinical research and medical education.

Education & Training

University of Tennessee Health Science CenterMD, 2011
University of TennesseeResidency, Internal Medicine, 2011 - 2014
University of Tennessee Health Science CenterMS, Biomedical Engineering, 2001 - 2004
Government Medical CollegeResidency, Medicine, 2000 - 2001
Dayanand Medical College and HospitalInternship, 1999 - 2000
Dayanand Medical College Class of 1999

Certifications & Licensure

TN State Medical License 2013 - 2026
American Board of Internal Medicine Internal Medicine

Awards, Honors, & Recognition

Member Graduate Student Association, University of Tennessee, 2003
Honor Council Representative University of Tennessee, 2002, 2003
College Captain and College Color Dayanand Medical College, 1997, 1999
Twice Silver Medallist Medallist at National Intermedical College Lawn Tennis Tournament, 1997, 1998
Students’ Sports Secretary Dayanand Medical College, 1997
Chief Organizer North Zone Intermedical College Sports Tournament, 1997
Ganesh Benzoplast merit Scholarship 1995
School Chairman’s award for excellent stage performance 1993, 1994
National Talent Search Scholarship Governmentof India, 1992
Head Boy and President Students Council
Top 5% of the class throughout school and college
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Publications & Presentations

PubMed

Safety of Baricitinib 4 mg for the Treatment of Moderate to Severe Rheumatoid Arthritis.
Tracey D Dabal, Matthew B. Haltom, Puja P Patel, Christine K Son, Kiran Joglekar
Southern Medical Journal. 2021-05-01
7 citations
Impact of a Novel Post-Discharge Transitions of Care Clinic on Hospital Readmissions
Michael Baldino, Anne Marie Bonaguro, Sean Burgwardt, Andrea Lombardi, Cagney Cristancho
Journal of the National Medical Association. 2021-04-01
A Case Report of Disseminated Blastomycosis With Thyroid Involvement in a Pregnant Patient.
Amit L Jain, Rahul Peravali, Hemnishil K. Marella, Reshma Premkumar, Ankur Seth
Cureus. 2020-06-08

Journal Articles

Probiotics ameliorate the hydrogen peroxide-inducedepithelial barrier disruption by a PKC- and MAP kinase-dependent mechanism.
Seth A, Yan F, Polk DB, Rao RK, Am J Physiol Gastrointest Liver Physiol
Lipopolysaccharide disrupts tight junctions in cholangiocyte monolayers by a c-Src-, TLR4-, and LBP-dependent mechanism.
Sheth P, Delos Santos N, Seth A, LaRusso NF, Rao RK, Am J Physiol Gastrointest Liver Physiol
L-Glutamine ameliorates acetaldehyde-induced increase in paracellular permeability in Caco-2 cell monolayer.
Seth A, Basuroy S, Sheth P, Rao RK, Am J Physiol Gastrointest Liver Physiol
Epidermal growth factor prevents acetaldehyde-induced paracellular permeability in Caco-2 cell monolayer.
Sheth P, Seth A, Thangavel M, Basuroy S, Rao RK, Alcohol Clin Exp Res
Recent Advances in Alcoholic Liver Disease I. Role of intestinal permeability and endotoxemia in alcoholic liver disease.
Rao RK, Seth A, Sheth P, Am J Physiol Gastrointest Liver Physiol
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Abstracts/Posters

C-Src Prevents Acetaldehyde-Induced Disruption Of Tight Junction, Contrary To its Requirement in the Disruption Of Tight Junction By Hydrogen Peroxide.
A. Seth, P. Sheth, R. Rao, Gastroenterology
Interaction of Protein Phosphatases, PP2A and PP1, with occludin delays the assembly of tight junctions in Caco-2 cells.
A. Seth, P. Sheth, R. Rao, Gastroenterology
MAP Kinase and PKC are independently involved in the mechanism of Epidermal Growth Factor (EGF)-mediated prevention of oxidative stress-induced disruption of tight j...
S. Basuroy, A. Seth, P. Sheth, R.K. Rao, Gastroenterology
PKC {epsilon} and PKC {beta} Are Required for EGF-Mediated Prevention Of Acetaldehyde-Induced Disruption Of Tight Junction.
A. Seth, P. Sheth, R. Rao, Gastroenterology
L-Glutamine prevents acetaldehyde-induced disruption of tight junctions (TJ) by preventing reorganization of actin cytoskeleton andchanges in phosphorylation of TJ pr...
A. Seth, S. Basuroy, P. Sheth, R. Rao, Gastroenterology
L-Glutamine-Mediated Prevention Of Acetaldehyde-Induced Disruption Of Tight Junction Involves C-Src, Metalloproteases (MMP) andLigand Binding Domain Of EGF Receptor (E...
A. Seth, R. Rao, Gastroenterology
Epidermal growth factor attenuates acetaldehyde-induced reorganization of actin cytoskeleton and disruption of tight junctions.
P. Sheth, A. Seth, S. Basuroy, R. Rao, Gastroenterology
Oxidative stress disrupts tight junctions by increasing the interaction of PP2A with occludin by Src-kinase dependent mechanism.
P. Sheth, A. Seth, S. Basuroy, R. Rao, Gastroenterology
Probiotic Lactobacillus GG secretory factors ameliorate hydrogen peroxide-induced disruption of intestinal epithelial tight junctions.
A. Seth, F. Yan, D. Brent Polk, R.K. Rao, Gastroenterology
Inhibition of JNK Activity Attenuates Osmotic Stress-Induced Disruption of Tight Junctions (TJ), and Reduced Expression of JNK1/2 Accelerates the Assembly.
TJ Geetha Samak, Takuya Suzuki, Ankur Seth, Parimal Sheth, Julia J. Hoy, Aditi Bhargava, R.K. Ra, DDW
Epidermal Growth Factor (EGF) Prevents Hydrogen Peroxide-Induced Disruption of Tight Junctions (TJ) in Cholangiocyte Monolayers.
S. Guntaka, A. Seth, N.F. LaRusso and R. K. Rao, DDW
Inhibition and Reduced Expression of PKCζAttenuates Phosphorylation of Occludin on Serine and Threonine, Resulting in the Disruption of Tight Junctions (TJ) and Delay ...
S. Jain, A. Seth and R. K. Rao, DDW
PKC {epsilon} and PKC {beta} Are Required for EGF-Mediated Prevention Of Acetaldehyde-Induced Disruption Of Tight Junction.
A. Seth, P. Sheth, R. Rao, DDW
L-Glutamine prevents acetaldehyde-induced disruption of tight junctions (TJ) by preventing reorganization of actin cytoskeleton andchanges in phosphorylation of TJ pro...
A. Seth, S. Basuroy, P. Sheth, R. Rao, DDW
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Lectures

Interaction of Protein Phosphatases, PP2A and PP1, with occludin delays the assembly of tight junctions in Caco-2 cells.
DDW
MAP Kinase and PKC are independently involved in the mechanism of Epidermal Growth Factor (EGF)-mediated prevention of oxidative stress-induced disruption of tight jun...
DDW
Regulated Interaction of PP2A and PP1 with OccludinDuring the Assembly of Tight Junctions Control Ser/Thr Phosphorylation of Occludin and the Assembly of Tight Junctions.
DDW
Phosphorylation of Y398 and Y402 in Occludin Prevents its Interaction with ZO-1 and Attenuates its Assembly into Tight Junctions (TJ).
DDW
EGF Activates PKCβI and PKCεby PLCγand Calcium-Dependent Mechanisms, and Prevents Acetaldehyde-Induced Disruption of Tight Junctions.
DDW
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Professional Memberships

American Medical Association - AMA
Member