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Alan Faden, MD, Neurology, Baltimore, MD

AlanI.FadenMDFACP, FAAN, FANA

Neurology Baltimore, MD

Professor and Associate Dean at University of Maryland School of Medicine

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  • Office

    20 Penn St
    HSF II, room S247
    Baltimore, MD 21230
    Phone+1 410-706-4205
    Fax+1 410-706-1639

Summary

  • Dr. Alan Faden is a neurologist in Baltimore, MD and is affiliated with University of Maryland Medical Center. He received his medical degree from University of Chicago Division of the Biological Sciences The Pritzker School of Medicine and has been in practice 46 years. He has expertise in neurotrauma, neuroimmunology and molecular neuroscience. . He has more than 400 publications and over 43,000 citations.

Education & Training

  • University of California (San Francisco)
    University of California (San Francisco)Residency, Neurology, 1972 - 1975
  • University of Pennsylvania Health System
    University of Pennsylvania Health SystemInternship, Internal Medicine, 1971 - 1972
  • University of Chicago Division of the Biological Sciences The Pritzker School of Medicine
    University of Chicago Division of the Biological Sciences The Pritzker School of MedicineClass of 1971

Certifications & Licensure

  • CA State Medical License
    CA State Medical License 1972 - Present
  • MD State Medical License
    MD State Medical License 1976 - 2026
  • DC State Medical License
    DC State Medical License 1991 - 2018
  • American Board of Psychiatry and Neurology Neurology

Awards, Honors, & Recognition

  • Distinguished Service Award University of Chicago, 2001
  • Exceptional Service Medal Uniformed Services University of the Health Sciences, 1984
  • American Society for Clinical Investigation 1981
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Publications & Presentations

PubMed

Press Mentions

  • Depletion and Repopulation of Microglia Could Be a Future Therapy for TBI
    Depletion and Repopulation of Microglia Could Be a Future Therapy for TBIFebruary 26th, 2020
  • UM School of Medicine's Shock Trauma and Anesthesiology Research (STAR) Center Announces Leadership Transition
    UM School of Medicine's Shock Trauma and Anesthesiology Research (STAR) Center Announces Leadership TransitionJanuary 27th, 2020
  • Microbleeds After Head Trauma Lead to Worse Neurological Outcomes
    Microbleeds After Head Trauma Lead to Worse Neurological OutcomesNovember 21st, 2019
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Grant Support

  • Microglial NOX2 drive pro-inflammatory responses and neurodegeneration after traumatic brain injury.National Institute of Neurological Disorders and Stroke2022–2027
  • Bidirectional Brain-Gut interactions Recovery, chronic neuroinflammation and neurodegeneration after Traumatic Brain Injury.National Institute of Neurological Disorders and Stroke2022–2027
  • Dementia Following Spinal Cord Injury: Mechanism and Therapeutic TargetingNational Institue of Neurologicl Disorders and Stroke2019–2024
  • Mechanism of Inflammatory Related Brain Dysfunction after Spinal Cord Injury.National Institute of Neurological Disorders and Stroke2019–2024
  • Reprogramming Microglial Epigenetic Pathways to Promote Cognitive Recovery after Brain Trauma.National Institue of Neurological Disorders and Stroke2019–2024
  • Spinal Mechanisms Underlying SCI-Induced Pain: Implications for Targeted Therapy.National Institute of Nursing Research2012–2023
  • miR-711 regulation of neuronal cell death after traumatic brain injury.NIH2016–2022
  • Effect of Hypobaria on Systemic Immune Function following Brain TraumaAir Foce2018–2021
  • Mechanisms of Autophagy-Lysosomal Pathway in Traumatic Brain InjuryNIH2015–2021
  • Role of miR-23a/27a in secondary injury after TBI.NIH2015–2021
  • mGluR5 inhibits microglial activation and neuronal cell death after TBINIH1998–2021
  • Biomarkers and Treatment of Hypobaria-Exacerbated TBIAir Force2016–2019
  • Role of Cell Cycle Pathways after Traumatic Brain InjuryNIH2006–2018
  • Center for the Genomics of PainNIH2012–2017
  • Prolonged Hypobaria during Aeromedical Evacuation and the Effects on TBIAir Force2011–2014
  • Mechanism and Modulation of Cell Death in Traumatic brain InjuryNIH2009–2014
  • Cell Cycle Pathways And Spinal Cord InjuryNational Institute Of Neurological Disorders And Stroke2010–2011
  • Combination Drug Treatment To Inhibit Multiple Cell Death Pathways After TBIEunice Kennedy Shriver National Institute Of Child Health &Human Development2009–2010
  • Cell Cycle Pathways And Spinal Cord InjuryNational Institute Of Neurological Disorders And Stroke2007–2010
  • Role Of Cell Cycle Pathways In Traumatic Brain InjuryNational Institute Of Neurological Disorders And Stroke2009
  • Mglur5 Inhibits Microglial Activation And Neuronal Cell Death After TBINational Institute Of Neurological Disorders And Stroke2009
  • Mechanisms And Modulation Of Cell Death In Traumatic Brain InjuryNational Institute Of Neurological Disorders And Stroke2009
  • Role Of Cell Cycle Pathways In Traumatic Brain InjuryNational Institute Of Neurological Disorders And Stroke2006–2009
  • Modulation Of Injury By Metabotropic Glutamate ReceptorsNational Institute Of Neurological Disorders And Stroke1999–2006
  • Apoptosis Induced By Traumatic Brain InjuryNational Institute Of Neurological Disorders And Stroke1998–2005
  • Core--Cellular Neuroscience FacilityEunice Kennedy Shriver National Institute Of Child Health &Human Development2001–2002
  • Multipontential Drug Treatment Strategies In NeurotramaNational Institute Of Neurological Disorders And Stroke2000–2002
  • Excitotoxins, Cellular Bioenergetics, And Brain InjuryNational Institute Of Neurological Disorders And Stroke1994
  • Excitotoxins, Cellular Bioenergetics And Brain InjuryNational Institute Of Neurological Disorders And Stroke1991–1993
  • Exitotoxins, Bioenergetics &Traumatic Brain InjuryNational Institute Of Neurological Disorders And Stroke1990–1991
  • Opioids, Opiate Receptors And Spinal Cord InjuryNational Institute Of Neurological Disorders And Stroke1987–1989