TritiaReinaYamasakiMDPhD

Neurology • Lexington, KY

Movement Disorder

Associate Professor with Tenure

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Office
740 South Limestone St
Dept of Neurology,J401
Lexington, KY 40513
Phone+1 859-323-5661

Fax+1 859-323-4607

Summary

Dr. Tritia Yamasaki is a neurologist in Lexington, KY and is affiliated with University of Kentucky Albert B. Chandler Hospital. She received her bachelors from Yale University, her MD and PhD degrees from University of California Irvine School of Medicine and did residency training in Neurology at UCLA and a Movement disorder fellowship at Washington University in St Louis. She has been in practice 8 years. She specializes in movement disorders and conducts research in Parkinson’s disease.

Education & Training

Washington University/B-JH/SLCH ConsortiumFellowship, Neuromuscular Medicine (Neurology), 2013 - 2015
UCLA David Geffen School of Medicine/UCLA Medical CenterResidency, Neurology, 2010 - 2013
University of California (Irvine)Residency, Internal Medicine, 2009 - 2010
University of California, Irvine, School of MedicineClass of 2009
Yale UniversityBS, Biology

Certifications & Licensure

CA State Medical License 2011 - Present
KY State Medical License 2015 - 2025
MO State Medical License 2013 - 2025
American Board of Psychiatry and Neurology Neurology

Publications & Presentations

PubMed

533 citations
Lipopolysaccharide-Induced Inflammation Exacerbates Tau Pathology by a Cyclin-Dependent Kinase 5-Mediated Pathway in a Transgenic Model of Alzheimer's Disease
Masashi Kitazawa, Salvatore Oddo, Tritia R. Yamasaki, Kim N. Green, Frank M. LaFerla
The Journal of Neuroscience. 2005-09-28
611 citations
Neural stem cells improve cognition via BDNF in a transgenic model of Alzheimer disease.
Mathew Blurton-Jones, Masashi Kitazawa, Hilda Martinez-Coria, Nicholas A. Castello, Franz-Josef Müller
Proceedings of the National Academy of Sciences of the United States of America. 2009-08-11
417 citations
Proteopathic tau seeding predicts tauopathy in vivo
Brandon B. Holmes, Jennifer L. Furman, Thomas E. Mahan, Tritia R. Yamasaki, Hilda Mirbaha
Proceedings of the National Academy of Sciences of the United States of America. 2014-10-14

Journal Articles

Parkinson’s Disease and Multiple System Atrophy Have Distinct Α-synuclein Seed Characteristics
Brandon B. Holmes, Tritia R. Yamasaki, Marc I. Diamond, Nigel J. Cairns, Paul T. Kotzbauer, The Journal of Biological Chemistry
Proteopathic tau seeding predicts tauopathy in vivo.
Holmes BB, Furman JL, Mahan TE, Yamasaki TR, Mirbaha H, Eades WC, Belaygorod L, Cairns NJ, Holtzman DM, Diamond MI., Proc Natl Acad Sci U S A
Cerebrospinal fluid biomarkers in clinical subtypes of early-onset Alzheimer's disease.
Teng E, Yamasaki TR, Tran M, Hsiao JJ, Sultzer DL, Mendez MF., Dement Geriatr Cogn Disord, 12/2013
Neural stem cells improve cognition via BDNF in a transgenic model of Alzheimer disease.
Blurton-Jones M, Kitazawa M, Martinez-Coria H, Castello NA, Müller FJ, Loring JF, Yamasaki TR, Poon WW, Green KN, LaFerla FM., Proc Natl Acad Sci U S A, 7/2009
Activation of cell cycle proteins in transgenic mice in response to neuronal loss but not amyloid-beta and tau pathology.
Lopes JP, Blurton-Jones M, Yamasaki TR, Agostinho P, LaFerla FM., J Alzheimers Dis, 2009
Neural stem cells improve memory in an inducible mouse model of neuronal loss.
Yamasaki TR, Blurton-Jones M, Morrissette DA, Kitazawa M, Oddo S, LaFerla FM., J Neurosci., 10/2007
Lipopolysaccharide-induced inflammation exacerbates tau pathology by a cyclin-dependent kinase 5-mediated pathway in a transgenic model of Alzheimer's disease.
Kitazawa M, Oddo S, Yamasaki TR, Green KN, LaFerla FM., J Neurosci, 9/2005
Microglia as a potential bridge between the amyloid beta-peptide and tau.
Kitazawa M, Yamasaki TR, LaFerla FM., Ann N Y Acad Sci, 12/2004
Inclusion body myositis-like phenotype induced by transgenic overexpression of beta APP in skeletal muscle.
Sugarman MC, Yamasaki TR, Oddo S, Echegoyen JC, Murphy MP, Golde TE, Jannatipour M, Leissring MA, LaFerla FM., Proc Natl Acad Sci U S A., 4/2002
Calsenilin reverses presenilin-mediated enhancement of calcium signaling.
Leissring MA, Yamasaki TR, Wasco W, Buxbaum JD, Parker I, LaFerla FM., Proc Natl Acad Sci U S A., 7/2000
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