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Office
20 York St
YNHH Childrens West Pavilion 2ND floor
New Haven, CT 06504Phone+1 203-785-4081Fax+1 203-785-6337
Summary
- Dr. Marie Egan is a pediatric pulmonologist in New Haven, CT and is affiliated with multiple hospitals in the area, including Greenwich Hospital and Yale-New Haven Hospital. She received her medical degree from Icahn School of Medicine at Mount Sinai and has been in practice 31 years. She specializes in cystic fibrosis pulmonology and pediatric bronchoscopy and is experienced in pneumonia, cystic fibrosis, chronic lung disease, flexible bronchoscopy, and pediatric bronchoscopy.
Education & Training
- Johns Hopkins UniversityFellowship, Pediatric Pulmonology, 1989 - 1992
- Johns Hopkins UniversityResidency, Pediatrics, 1986 - 1988
- Icahn School of Medicine at Mount SinaiClass of 1986
- University of PennsylvaniaB.A., Biology
Certifications & Licensure
- CT State Medical License 1992 - 2025
- MD State Medical License 1990 - 1992
- American Board of Pediatrics Pediatrics
- American Board of Pediatrics Pediatric Pulmonology
Clinical Trials
- G551D Observational Study- Expanded to Additional Genotypes and Extended for Long Therm Follow up (GOAL-e2) Start of enrollment: 2014 Jan 01
- Microbiome Acquisition and Progression of Inflammation and Airway Disease in Infants and Children With Cystic Fibrosis Start of enrollment: 2012 Aug 01
- OPTIMIZing Treatment for Early Pseudomonas Aeruginosa Infection in Cystic Fibrosis Start of enrollment: 2014 Jun 01
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Publications & Presentations
PubMed
- Ezrin drives adaptation of monocytes to the inflamed lung microenvironment.Ravindra Gudneppanavar, Caterina Di Pietro, Hasan H Öz, Ping-Xia Zhang, Ee-Chun Cheng
Cell Death & Disease. 2024-11-29 - 1 citationsCCR2+ monocytes are dispensable to resolve acute pulmonary Pseudomonas aeruginosa infections in WT and Cystic Fibrosis mice.Hasan H Öz, Cassia L Braga, Ravindra Gudneppanavar, Caterina Di Pietro, Pamela H Huang
Journal of Leukocyte Biology. 2024-10-04 - Localization and function of humanized F508del-CFTR in mouse intestine following activation of serum glucocorticoid kinase 1 and Trikafta.Parinaz Dastoor, Caroline Muiler, Alannah Garrison, Marie Egan, Diego Carlos Dos Reis
European Journal of Pharmacology. 2024-09-05
Press Mentions
- 2023 ‘Top Doctors’ List Features Over 250 Yale Medicine PhysiciansMay 3rd, 2023
- Yale Named Top Ten Center of Biomedical Research by the Hartwell FoundationJune 24th, 2020
- 5,000 Expected to Attend Cystic Fibrosis Conference, NACFC 2019, in NashvilleOctober 7th, 2019
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Grant Support
- The Effects Of CFTR Dysfunction On Bone FormationNational Institute Of Arthritis And Musculoskeletal And Skin Diseases2011–2012
- CFTR And The Immune ResponseNational Heart, Lung, And Blood Institute2010–2012
- Etiology Of Cystic Fibrosis Bone DiseaseNational Institute Of Arthritis And Musculoskeletal And Skin Diseases2009
- CFTR And Ion Channels: Mechanism Of InteractionNational Institute Of Diabetes And Digestive And Kidney Diseases2004–2006
- CFTR Regulation Of Ion ChannelsNational Institute Of Diabetes And Digestive And Kidney Diseases1998–2002
- Regulation Of Airway Epithelial Chloride ChannelsNational Heart, Lung, And Blood Institute1994–1997
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